Increased soft tissue deposits around the upper airway will reduce the size of the upper airway
Predisposing risk factors of OSA
There are a number of factors which influence an individual’s likelihood of developing OSA. Considering the development of upper airway occlusion during sleep, it is logical to assume that anything reducing the size of the upper airway will increase the likelihood of developing obstructive sleep apnoea.
Increased soft tissue deposits around the upper airway will reduce the size of the upper airway
There is a higher prevalence of OSA in males, which is partially due to physiological differences in gender, but also to progesterone levels. This gender difference neutralises when women reach menopause
Resulting in muscular and neurological loss of muscle tone of the upper airway
Temporary decrease in muscle tone caused by muscle relaxants, sedatives, anesthetics and opioid analgesics
Caused by traumatic brain injury, neuromuscular disorders, or other disorders
Structural features that reduce the size of the upper airway, including a receding jaw, large neck circumference, nasal congestion, brachycephaly. Characteristic features of genetic craniofacial syndromes may involve the nose, mouth, jaw and resting muscle tone. For example, in Down Syndrome there are some characteristic features of the syndrome that predispose to OSA, including an enlarged tongue, low muscle tone and a narrow nasopharynx
Certain irritants can cause inflammation of the upper airway, leading to narrowing. These include smoking and air pollution
Those with a family history of OSA are more likely to develop OSA
The upper airway have collections of lymphoid tissue, or tonsils, which act as a line of defence again inhaled and ingested pathogens. When these become enlarged (adenotonsillar hyperplasia) or inflamed (tonsillitis) this can occlude the upper airway
Certain conditions directly cause OSA, including acromegaly, androgen therapy, hypothyroidism and polycystic ovarian syndrome
However, some individuals with an apparently normal upper airway and no other risk factors or predictors of the condition can indeed develop OSA.
There are a number of factors which influence an individual’s likelihood of developing OSA. Considering the development of upper airway occlusion during sleep, it is logical to assume that anything reducing the size of the upper airway will increase the likelihood of developing obstructive sleep apnoea.
Increased soft tissue deposits around the upper airway will reduce the size of the upper airway
There is a higher prevalence of OSA in males, which is partially due to physiological differences in gender, but also to progesterone levels. This gender difference neutralises when women reach menopause
Resulting in muscular and neurological loss of muscle tone of the upper airway
Temporary decrease in muscle tone caused by muscle relaxants, sedatives, anesthetics and opioid analgesics
Caused by traumatic brain injury, neuromuscular disorders, or other disorders
Structural features that reduce the size of the upper airway, including a receding jaw, large neck circumference, nasal congestion, brachycephaly. Characteristic features of genetic craniofacial syndromes may involve the nose, mouth, jaw and resting muscle tone. For example, in Down Syndrome there are some characteristic features of the syndrome that predispose to OSA, including an enlarged tongue, low muscle tone and a narrow nasopharynx
Certain irritants can cause inflammation of the upper airway, leading to narrowing. These include smoking and air pollution
Those with a family history of OSA are more likely to develop OSA
The upper airway have collections of lymphoid tissue, or tonsils, which act as a line of defence again inhaled and ingested pathogens. When these become enlarged (adenotonsillar hyperplasia) or inflamed (tonsillitis) this can occlude the upper airway
Certain conditions directly cause OSA, including acromegaly, androgen therapy, hypothyroidism and polycystic ovarian syndrome
However, some individuals with an apparently normal upper airway and no other risk factors or predictors of the condition can indeed develop OSA.